I used chatgpt to simplify the post further below
Central poststroke pain (CPSP) is a type of nerve pain that can occur after a stroke. The exact cause isn’t fully understood, but one theory is that damage from the stroke reduces the number of inhibitory (pain-blocking) neurons in the spinothalamic tract. This loss of inhibition may cause certain thalamic neurons to become overactive and fire on their own, creating false pain signals that the brain interprets as real pain.
This was the original text, I was just about to say this myself ! 😁
Central poststroke pain (CPSP) is a neuropathic pain disorder, the underlying mechanisms of which are not well understood. It has been suggested that stroke-associated loss of inhibitory neurons in the spinothalamic tract causes disinhibition of thalamic neurons, which autonomously generate ectopic nociceptive action potentials responsible for the pain experience. We hypothesized that CPSP is a result of misinterpretation of afferent sensory input by the sensitized neurons within the brain, rather than generated spontaneously by the damaged central nervous system (CNS) neurons